This research explores CNS pathways influencing intraocular pressure, suggesting implications for ocular health.
Efferent and afferent pathways by which the central nervous system (CNS) may control intraocular pressure (IOP) are considered. As experience with pharmaceutical agents has suggested, the targets for autonomie influence on ocular tension are both aqueous formation and resistance to outflow. The afferent input to the brain must be served by sensory fibers which are sensitive to intraocular pressure levels or which respond to changes in intraocular pressure. The location and physiology of such receptors is discussed.
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Jan P.G. Bergmanson (1982) studied this question.
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