Renal resilience represents the functional reserve capacity of the kidneys, which becomes measurable following unilateral nephrectomy as change in single-kidney glomerular filtration rate (ΔskGFR). While the causes and consequences of inter-individual differences in renal resilience are incompletely understood, dysregulation of metabolic processes is conceivably involved. The aim of this research was to investigate whether plasma ketone bodies, as potential markers of lipid and energy metabolism, are associated with renal resilience in living kidney donors.
Nizamoglu et al. (2026) studied this question.