Experimental blockade of interleukin-33 reduces synovial inflammation and joint damage in rheumatoid arthritis, suggesting a viable treatment option.
IL-33 blockade effectively mitigates synovial inflammation and joint destruction by suppressing the proliferation, migration, and invasion of RA-FLSs while promoting their apoptosis. Targeting IL-33 may represent a promising strategy for the treatment of rheumatoid arthritis.
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Feng et al. (2026) studied this question.
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