Study shows that NLRP3 inflammasome contributes to infarct size in myocardial fibroblasts after MI, indicating potential therapeutic targets.
This study shows that the NLRP3 inflammasome is up-regulated in myocardial fibroblasts post-MI, and may be a significant contributor to infarct size development during ischaemia-reperfusion.
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Sandanger et al. (2013) studied this question.
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