Metabolic engineering enhances antitumor activity in solid tumors, suggesting a potent strategy for CAR-Ms.
Metabolic engineering via SLC38A2 restored glutamine fitness and enhanced the antitumor activity of HER2-targeted CAR-Ms, thus providing a promising strategy to boost CAR-M-mediated tumor suppression in solid tumors.
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Liu et al. (2026) studied this question.
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