Immunohistological pointers to a possible role for excessive cathelicidin (LL‐37) expression by apocrine sweat glands in the pathogenesis of hidradenitis suppurativa/acne inversa

Our observations raise the question as to whether excessive secretion of AMPs by the skin, in particular by the apocrine sweat glands, distal HF epithelium, and epidermis, may attract inflammation and thus facilitate or promote HS development.