Key result
O-GlcNAc signaling provides endogenous cardioprotection, potentially by directly modifying mitochondrial proteins.
Why the study?
O-GlcNAc signaling is a unique endogenous mechanism of cardioprotection that may involve modification of mitochondrial proteins critical for survival.
O-GlcNAc signaling represents a potential endogenous mechanism for cardioprotection via mitochondrial protein modification.
O-GlcNAc signaling represents a unique endogenously recruitable mechanism of cardioprotection that may involve direct modification of mitochondrial proteins critical for survival such as voltage-dependent anion channel.
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Jones et al. (2008) studied this question. O-GlcNAc signaling acts as an endogenous mechanism of cardioprotection that may involve direct modification of mitochondrial proteins such as the voltage-dependent anion channel.
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