Preclinical study demonstrates sympathetic activation of hematopoietic stem cells in ischemic stroke, indicating a neural mechanism driving post-stroke inflammation.
Ischemic stroke activates hematopoietic stem cells via increased sympathetic tone, leading to a myeloid bias of hematopoiesis and higher bone marrow output of inflammatory Ly6C(high) monocytes and neutrophils.
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Courties et al. (2014) studied this question.
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