Population
Patients with bipolar disorder and schizophrenia
Design
Other
Authors
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Hypothesis-generating for NCAM1 in psychosis; leaves open whether alternative splicing drives shared bipolar-schizophrenia risk.
SNPs within NCAM1 may contribute to the risk of bipolar disorder and schizophrenia through alternative splicing.
Atz et al. (2007) studied this question.
Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context: