Patients with type 2 diabetes and normal cardiac function exhibit impaired myocardial and skeletal muscle energy metabolism linked to circulating metabolic substrates.
Highlights potential early metabolic impairment in T2D despite normal function; leaves open whether substrate modulation prevents progression.
Type 2 diabetic patients with apparently normal cardiac function have impaired myocardial and skeletal muscle energy metabolism related to changes in circulating metabolic substrates.
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Scheuermann‐Freestone et al. (2003) studied this question.
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