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TRPM3 activity is impaired in CFS/ME patients suggesting changes in intracellular Ca²⁺ concentration, which may impact NK cellular functions. This investigation further helps to understand the intracellular-mediated roles in NK cells and confirm the potential role of TRPM3 ion channels in the aetiology and pathomechanism of CFS/ME.
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Cabanas et al. (2018) studied this question.
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