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March 1, 2021Journal for ImmunoTherapy of CancerOpen Access

Tumor-associated neutrophils and macrophages interaction contributes to intrahepatic cholangiocarcinoma progression by activating STAT3

Zheng‐Jun Zhou Pengcheng Wang Rongqi Sun Jia Li Zhiqiang Hu Haoyang Xin Chu‐Bin Luo Jian Zhou Jia Fan Shao‐Lai Zhou

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TANs and TAMs interact to promote ICC progression by activating STAT3.

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Zhou et al. (2021) studied this question.

synapsesocial.com/papers/69db5603f7e0c66ced835a02 https://doi.org/10.1136/jitc-2020-001946

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Macrophages display spatiotemporal specificity in intrahepatic cholangiocarcinoma and drive tumour progression via OSM and THBS12025
2DEFB1 + Cancer Cell– IFI30 + Macrophage Crosstalk Drives Immunosuppressive Niche in Intrahepatic CholangiocarcinomaOriginal Research · 2026
3A Multi-omics Investigation Identifies TACC3 as a Driver of Immunosuppression in Intrahepatic Cholangiocarcinoma via Activation of the STAT3-PD-L1 AxisOriginal Research · 2026
4Tumor-Associated Neutrophils Recruit Macrophages and T-Regulatory Cells to Promote Progression of Hepatocellular Carcinoma and Resistance to SorafenibOriginal Research · 2016 · 854 citations
5The Inflammatory Microenvironment in Hepatocellular Carcinoma: A Pivotal Role for Tumor-Associated MacrophagesOriginal Research · 2012 · 408 citations

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