Experimental study demonstrates rhFGF21 promotes functional recovery by dampening neuroinflammation in ischemic stroke models, suggesting therapeutic utility.
rhFGF21 treatment promoted functional recovery in experimental stroke by modulating microglia/macrophage-mediated neuroinflammation via the NF-κB and PPAR-γ signaling pathways, making it a potential anti-inflammatory agent for stroke treatment.
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Wang et al. (2020) studied this question.
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