What question did this study set out to answer?

This research aims to investigate how lipid remodeling and phosphatidic acid signaling impact stress responses in Panax notoginseng seeds at low temperatures.

April 14, 2026Open Access

Lipid remodeling and phosphatidic acid signaling maintain the vigor of recalcitrant Panax notoginseng seeds under low temperature

Key Points

This research aims to investigate how lipid remodeling and phosphatidic acid signaling impact stress responses in Panax notoginseng seeds at low temperatures.
Analyzed membrane-lipid microenvironment changes under low temperatures of 0 °C and 4 °C.
Investigated the signaling pathways involving ABA and transcription factors.
Examined the effects of lipid changes on seed vigor.
Recalcitrant seeds show insensitivity to low temperatures.
Lipid microenvironment changes trigger ABA signaling pathways.
Coordination of stress responses involves NAC and DREB transcription factors.

Abstract

In conclusion, recalcitrant P. notoginseng seeds appear insensitive to low temperature at 0 °C and 4 °C. Changes in the membrane-lipid microenvironment initiate an integrated signaling mechanism that transduces signals via the core ABA pathway. This integrated mechanism ultimately converges on NAM, ATAF1/2, and CUC2 (NAC) transcription factors and dehydration-responsive element-binding protein (DREB) transcription factors to coordinate stress responses.

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Lipid remodeling and phosphatidic acid signaling maintain the vigor of recalcitrant Panax notoginseng seeds under low temperature

Key Points

Abstract

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