Why the study?
Does the presence of perivascular adipose tissue improve endothelial function and insulin-induced vasodilatation in hypercholesterolemic LDL receptor-deficient mice?
Does the presence of perivascular adipose tissue improve endothelial function and insulin-induced vasodilatation in hypercholesterolemic LDL receptor-deficient mice?
Elevated eNOS-derived NO production in aortic PVAT acts as a compensatory mechanism to restore endothelial function and insulin-induced vasodilation in hypercholesterolemic LDLr-deficient mice.
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Suggests compensatory PVAT role in murine hypercholesterolemia; hypothesis-generating and requires human validation.
Baltieri et al. (2018) studied this question.
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