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This research uncovers NLRP3 inflammasome's role in coronary artery lesions in Kawasaki Disease, suggesting a therapeutic target.
NLRP3 inflammasome activation during the progression of KD can cause coronary endothelial injury by inducing inflammatory imbalance, endothelial cells pyroptosis, and glycocalyx injury. Targeting NLRP3 represents a promising therapeutic strategy.
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Zheng et al. (2026) studied this question.
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