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Multicenter studies reveal that lower EMMPRIN levels in uterine ligaments contribute to pelvic organ prolapse, suggesting a potential mechanism.
EMMPRIN levels in the uterosacral ligament, cardinal ligament and ligamentum rotundum were significantly lower in patients with POP. EMMPRIN deficiency may be one mechanism underlying the development of POP, but multicenter studies with larger patient numbers are needed.
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Yildiz et al. (2026) studied this question.
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