This theoretical paper explores platelet dysfunction and non-coding RNA's role in systemic lupus erythematosus, suggesting new avenues for research.
This theoretical paper presents a hypothetical framework exploring the role of platelet dysfunction, non-coding RNA dysregulation, and systemic repair failure in systemic lupus erythematosus (SLE). It proposes that insufficient systemic repair capacity, modulated by platelet function and ncRNA expression, may contribute to chronic inflammation and immune dysregulation in a subset of SLE patients. The model is complementary to traditional autoimmune theories and intended to stimulate further exploratory research.
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FOO SENG ANG (2026) studied this question.
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