Inhibiting Nrf2/HO-1 signaling reduces lung inflammation caused by gasoline engine exhaust, suggesting new treatment targets.
Inhibiting the Nrf2/HO-1 signaling cascade attenuates the pro-inflammatory response induced by GEE in lung epithelial cells following ALI exposure. The Nrf2/HO-1 pathway appears to be a critical regulator of GEE-induced pulmonary inflammation, highlighting its potential as a therapeutic target.
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Qu et al. (2026) studied this question.
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