Collectively, the anti-inflammatory mechanisms of EA might be related to the decrease in the level of MDA, iNOS, and COX-2 in the edema paw via the suppression of pro-inflammatory cytokines (TNFα, IL1 β), NO and PGE2 overproduction.
No takes yet. Share an insight, caveat, or question.
Ghorbanzadeh et al. (2015) studied this question.
Synapse has enriched 5 related papers. Consider them for comparative context: