Translational study reveals interleukin-7 drives plaque instability in coronary artery disease, highlighting critical interactions among immune cells.
Our findings suggest a role for IL-7-driven inflammation in atherogenesis and the promotion of clinical instability in coronary artery disease involving interactions between platelets, monocytes, and chemokines.
No takes yet. Share an insight, caveat, or question.
Damås et al. (2003) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: