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April 30, 2026International Journal of Molecular Sciences2 citationsOpen Access

Protective Effects of Astragalin Against Acute Ultraviolet B-Induced Photodamage in HaCaT Cells and Mouse Skin

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PTPei TangYWYue WangXHXuanhao Huang

Key Points

  • To investigate the protective effects of astragalin against UVB-induced skin damage.
  • In vitro examination using HaCaT keratinocytes to assess cytotoxicity and apoptosis
  • In vivo study with topical application of astragalin on Kunming mice
  • Analysis of reactive oxygen species and antioxidant activity
  • Assessment of proinflammatory cytokines and signaling pathways
  • Astragalin mitigated UVB-induced cytotoxicity and apoptosis in HaCaT cells
  • Topical astragalin application reduced erythema and epidermal hyperplasia in mouse skin
  • It decreased reactive oxygen species levels and enhanced antioxidant enzyme activity
  • AST suppressed proinflammatory cytokines via inhibiting the TLR4/NF-κB pathway

Abstract

Astragalin (AST), a natural flavonoid found in various plants, possesses antioxidant and anti-inflammatory properties. However, its protective efficacy against ultraviolet B (UVB)-induced cutaneous damage remains unclear. This study investigated the photoprotective effects of AST against UVB-induced photodamage using HaCaT keratinocytes and Kunming mice. In vitro, AST mitigated UVB-induced cytotoxicity and apoptosis in HaCaT cells. In vivo, topical application of AST attenuated UVB-induced erythema, epidermal hyperplasia, and collagen degradation in mouse skin. Additionally, AST reduced reactive oxygen species accumulation and enhanced antioxidant enzyme activity via activation of the Keap1/Nrf2 pathway. Furthermore, AST suppressed the expression of proinflammatory cytokines by inhibiting the TLR4/NF-κB signaling pathway. These findings demonstrate the photoprotective properties of AST and support its potential as a natural therapeutic agent for preventing UVB-induced skin damage.

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Cite This Study

Tang et al. (2026) studied this question.

synapsesocial.com/papers/69f2a4da8c0f03fd67763fe3https://doi.org/10.3390/ijms27093839
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