Shows erythroferrone from osteoblasts enhances RBC recovery during stress erythropoiesis, indicating a key regulatory mechanism.
mutants showed erythroid lineage proliferation and enhanced EPO responsiveness in a BMP4-dependent manner. Taken together, we posit that ERFE loss specifically from osteoblasts enhances RBC recovery during stress erythropoiesis-defining mechanisms of regulation in the crosstalk between osteoblasts and erythroblasts.
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Na-Phatthalung et al. (2026) studied this question.
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