Why the study?
Do slow conduction and underlying pathomorphologic changes at the RVOT contribute to the initiation of VF by programmed ventricular stimulation in patients with Brugada syndrome?
Do slow conduction and underlying pathomorphologic changes at the RVOT contribute to the initiation of VF by programmed ventricular stimulation in patients with Brugada syndrome?
Slow conduction at the RVOT, associated with pathomorphologic changes such as fibrosis and fatty infiltration, may contribute to the induction of ventricular fibrillation in Brugada syndrome.
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May implicate RVOT substrate in Brugada VF induction; hypothesis-generating from Level 5 data and should not change practice.
Ohkubo et al. (2010) studied this question.
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