January 1, 2018TheranosticsOpen Access

MicroRNA-378 suppresses myocardial fibrosis through a paracrine mechanism at the early stage of cardiac hypertrophy following mechanical stress.

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Abstract

: miR-378 is secreted from cardiomyocytes following mechanical stress and acts as an inhibitor of excessive cardiac fibrosis through a paracrine mechanism.

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Cite This Study

Yuan et al. (2018) studied this question.

synapsesocial.com/papers/69ff94444716aad0cc856558 https://doi.org/10.7150/thno.22878

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Abstract

: miR-378 is secreted from cardiomyocytes following mechanical stress and acts as an inhibitor of excessive cardiac fibrosis through a paracrine mechanism.

MicroRNA-378 suppresses myocardial fibrosis through a paracrine mechanism at the early stage of cardiac hypertrophy following mechanical stress.

Abstract

Discussion

Cite This Study

MicroRNA-378 suppresses myocardial fibrosis through a paracrine mechanism at the early stage of cardiac hypertrophy following mechanical stress.

Abstract

Discussion

Cite This Study

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