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November 1, 2019Frontiers in Molecular NeuroscienceOpen Access

Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

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Authors

LMLuke McAlaryUniversity of WollongongSPSteven S. PlotkinUniversity of British ColumbiaJYJustin J. YerburyUniversity of Wollongong

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McAlary et al. (2019) studied this question.

synapsesocial.com/papers/6a0190cf950a93c470d8ae4ehttps://doi.org/10.3389/fnmol.2019.00262
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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Induction of Amyloid Fibrils by the C-Terminal Fragments of TDP-43 in Amyotrophic Lateral Sclerosis2010 · 147 citations
  2. 2The Truncated C-terminal RNA Recognition Motif of TDP-43 Protein Plays a Key Role in Forming Proteinaceous Aggregates2013 · 118 citations
  3. 3Intranuclear Aggregation of Mutant FUS/TLS as a Molecular Pathomechanism of Amyotrophic Lateral Sclerosis2013 · 168 citations