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July 31, 2008Proceedings of the National Academy of SciencesOpen Access

DNA polymorphisms at the BCL11A , HBS1L-MYB , and β- globin loci associate with fetal hemoglobin levels and pain crises in sickle cell disease

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GLGuillaume LettreMontreal Heart InstituteVSVijay G. SankaranBroad InstituteMBMarcos BezerraUniversidade Federal de Pernambuco

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Lettre et al. (2008) studied this question.

synapsesocial.com/papers/6a035593bc3ffe278e6564fahttps://doi.org/10.1073/pnas.0804799105
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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The acute chest syndrome in sickle cell disease: incidence and risk factors. The Cooperative Study of Sickle Cell Disease1994 · 737 citations
  2. 2Intergenic variants of <i>HBS1L-MYB</i> are responsible for a major quantitative trait locus on chromosome 6q23 influencing fetal hemoglobin levels in adults2007 · 325 citations
  3. 3The BCL11 gene family: involvement of BCL11A in lymphoid malignancies2001 · 318 citations