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May 13, 2026Arthritis & Rheumatology2 citations

CD177+neutrophils promote endothelial senescence through extracellular traps in Behçet's Disease

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XWXiaoou WangXYX YuXWXun Wang

Key Points

  • This research aims to investigate the role of PMN dysregulation and extracellular traps in promoting endothelial senescence in Behçet's disease.
  • Assessed PMN-derived NETs and their components
  • Analyzed the effects of Histone 3.1 on VECs
  • Evaluated the role of TLR4 signaling and the impact of senolytics
  • PMN-derived NETs contained elevated levels of Histone 3.1
  • Histone 3.1 induced senescence, SASP, and PMN chemotaxis in VECs via TLR4 signaling
  • Senolytics attenuated the effects of H3.1 on VECs

Abstract

OBJECTIVE: PMN dysregulation on endothelial cells in Behçet's disease (BD), a chronic systemic vasculitis characterized by polymorphonuclear neutrophil (PMN) activation and endothelial dysfunction. METHODS: PMN-derived NETs were assessed. RESULTS: PMN-derived NETs contained more Histone 3.1 (H3.1). H3.1 induced senescence, SASP and PMN chemotaxis in VECs via TLR4 signaling, which was attenuated by senolytics. CONCLUSION: PMN in BD patients promote senescence and SASP in VECs through secreting H3.1-enriched NETs, which recruit PMN and collectively contribute to vasculitis in BD.

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Cite This Study

Wang et al. (2026) studied this question.

synapsesocial.com/papers/6a04147679e20c90b4444607https://doi.org/10.1002/art.70217
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