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AIMS: mice with pressure overload. METHODS AND RESULTS: transport and mitochondrial ROS generation, and prevented VCAM-1/ICAM-1 upregulation and EC dysfunction, eventually restrained atherosclerotic lesions development. CONCLUSION: mice. Nogo-B may hold the promise to be a common therapeutic target in the setting of hypertension.
Zhang et al. (Sat,) studied this question.