Key points are not available for this paper at this time.
, RvD1和PD1减少了多发性硬化症来源的单核细胞活化和细胞因子产生,抑制了炎症引起的血脑屏障功能障碍和单核细胞跨内皮迁移。总的来说,这些发现揭示了多发性硬化症中分解途径的外周缺陷,提示促分解脂质介质作为新的诊断生物标志物和潜在安全的治疗药物。
Kooij等(周四)研究了这个问题。
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: