Key result
Restoring sinus rhythm and optimal drug therapy normalizes LVEF in a tachycardia-induced cardiomyopathy case.
Why the study?
The article addresses the current issue of the etiology, diagnostic criteria, and treatment of cardiomyopathy resulting from persistent tachycardia.
Does restoration of sinus rhythm and optimal drug therapy improve cardiac function in a patient with tachycardia-induced cardiomyopathy?
Case Report (n=1)
Does restoration of sinus rhythm and optimal drug therapy improve cardiac function in a patient with tachycardia-induced cardiomyopathy?
Restoration of sinus rhythm in tachycardia-induced cardiomyopathy can lead to regression of symptoms and normalization of left ventricular function.
May support reversibility of tachycardia-induced cardiomyopathy with rhythm restoration; hypothesis-generating and requires prospective validation before practice change.
The article highlights current issue of the etiology of cardiomyopathy resulting from persistent tachycardia. Clinical studies devoted to the diagnosis and treatment of tachycardia-induced cardiomyopathy and the criteria for its diagnosis are presented. The article presents a clinical case report of a 48-year-old patient who developed cardiomyopathy against the background of long-term persistent tachysystolic atrial fibrillation. After the restoration of the sinus rhythm against the background of optimal drug therapy, the clinical signs of cardiomyopathy regressed, the size of the heart chambers and the contractile function of the myocardium of the left ventricle of the heart returned to normal.
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Steklov et al. (2022) conducted a case report in Tachycardia-induced cardiomyopathy (n=1). Restoration of sinus rhythm and optimal drug therapy was evaluated on Regression of clinical signs of cardiomyopathy, size of heart chambers, and contractile function of the left ventricle. Restoration of sinus rhythm and optimal drug therapy in a 48-year-old patient with tachycardia-induced cardiomyopathy resulted in the normalization of left ventricular contractile function.
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