Key result
Endurance athletes exhibited greater peak exercise right ventricular end-systolic wall stress than nonathletes (340 vs 266 kdyn·cm, P=0.028), driving disproportionate right ventricular remodeling.
Why the study?
Does intense endurance exercise cause disproportionate right ventricular load and remodeling compared to nonathletes?
Cross-Sectional (n=53)
Does intense endurance exercise cause disproportionate right ventricular load and remodeling compared to nonathletes?
Absolute Event Rate: 340% vs 266%
p-value: p=0.028
Intense endurance exercise places a disproportionate hemodynamic load on the right ventricle compared to the left ventricle, which may drive the greater right ventricular remodeling observed in athletes.
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Highlights potential mechanism for RV remodeling in endurance athletes; hypothesis-generating and should not yet alter practice.
Gerche et al. (2010) conducted a cross-sectional in Endurance athletes (n=53). Endurance athletic training vs. Nonathletes was evaluated on Peak exercise right ventricular end-systolic wall stress (RVES-σ) in kdyn·cm (p=0.028). Endurance athletes exhibited greater peak exercise right ventricular end-systolic wall stress than nonathletes (340 vs 266 kdyn·cm, P=0.028), driving disproportionate right ventricular remodeling.
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