Key result
The hemostatic system accelerates plaque vulnerability to thrombosis in experimental models, although clinical evidence supporting this effect remains weak.
Design
Review
Weak clinical evidence precludes practice change; leaves open whether hemostasis modifies human plaque thrombosis.
Experimental data suggest that the hemostatic system can accelerate plaque vulnerability to thrombosis in vivo. However, clinical support for these findings remains weak. This comprehensive summary reviews the evidence.
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Borissoff et al. (2011) studied this question. The hemostatic system accelerates plaque vulnerability to thrombosis in experimental models, although clinical evidence supporting this effect remains weak.
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