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Shows caffeine protects the retina from cell death during ischemia, suggesting new treatment options.
receptor antagonism and inhibition of glutamate excitotoxicity, rather than by activating canonical survival signaling pathways. This supports the potential use of caffeine as a rapid neuroprotective agent during acute ischemic events in the immature central nervous system.
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Nascimento et al. (2026) studied this question.
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