Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
September 10, 2011BloodOpen Access

Loss of the wild-type allele contributes to myeloid expansion and disease aggressiveness in FLT3/ITD knockin mice

View Full Paper
Ask AI
Bookmark
Share

Authors

LLLi LiThe University of Texas MD Anderson Cancer CenterEBEmily BaileyJohns Hopkins UniversitySGSarah GreenblattGenomics Institute of the Novartis Research Foundation

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Li et al. (2011) studied this question.

synapsesocial.com/papers/6a0879c1ab15ea61dee8df8dhttps://doi.org/10.1182/blood-2011-01-328096
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Identification of a lineage of multipotent hematopoietic progenitors1997 · 484 citations
  2. 2GATA-1 reprograms avian myelomonocytic cell lines into eosinophils, thromboblasts, and erythroblasts.1995 · 431 citations
  3. 3Targeted disruption of the flk2/flt3 gene leads to deficiencies in primitive hematopoietic progenitors1995 · 595 citations
  4. 4Internal tandem duplication mutation of FLT3 blocks myeloid differentiation through suppression of C/EBPα expression2003 · 157 citations