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) exhibited accelerated leaf senescence based on chlorophyll a/b ratio measurements and photochemical analyses. Photosynthetic complex formation was normal in all STN7 target protein loss-of-function mutants, indicating the absence of feedback regulation upstream of STN7 during photosynthetic complex assembly. Overall, the data reveal that STN7 is a major kinase component in signaling cascades that link photosynthetic complex formation and leaf senescence.
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Lee et al. (2025) studied this question.
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