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June 1, 1952Circulation213 citations

Dynamic Responses of the Right Ventricle following Extensive Damage by Cauterization

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AKAbraham Kagan

Key Result

Extensive damage to the right ventricular walls by cauterization caused only slight changes in venous and arterial pressures and did not produce right heart failure.

Structured PICO

Does extensive damage by cauterization of the right ventricular walls cause right heart failure and venous congestion?

P
Population
Preclinical model (unspecified in abstract) undergoing major destruction of the right ventricular walls
I
Intervention
Extensive damage by cauterization
O
Outcome
Changes in venous and arterial pressures and compensatory reaction to stresssurrogate

Extensive cauterization damage to the right ventricle does not produce right heart failure or venous congestion, suggesting independent regulation and enigmatic compensatory mechanisms.

Abstract

Previous observations that major destruction of the right ventricular walls causes but slight changes in venous and arterial pressures were confirmed. Furthermore, additional observations indicated that the compensatory reaction of the damaged ventricle to stress is diminished surprisingly little. The results are considered evidence that the cauterization procedure and others of a similar nature do not produce right heart failure and, therefore, would not be expected to cause venous congestion. The subsidiary muscular mechanism which maintains the right ventricular pump remains enigmatic; but evidence is submitted that the damaged right ventricle is regulated independently of the left.

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Cite This Study

Abraham Kagan (1952) studied Right ventricular damage. Cauterization of right ventricular walls was evaluated on Changes in venous and arterial pressures and compensatory reaction to stress. Extensive damage to the right ventricular walls by cauterization caused only slight changes in venous and arterial pressures and did not produce right heart failure.

synapsesocial.com/papers/6a09064729af591ab7017299https://doi.org/10.1161/01.cir.5.6.816
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