Key result
Acute changes in cardiac output and ejection fraction induced by dobutamine infusion and CABG did not result in significant alterations in the anatomic aortic valve area determined by transesophageal echocardiography using direct planimetry.
Why the study?
Do acute changes in cardiac output and ejection fraction alter the anatomic aortic valve area determined by TEE using direct planimetry in patients undergoing CABG?
Observational (n=17)
No
Do acute changes in cardiac output and ejection fraction alter the anatomic aortic valve area determined by TEE using direct planimetry in patients undergoing CABG?
Anatomic aortic valve area determined by TEE using direct planimetry remains constant despite acute changes in cardiac output and ejection fraction, making it a reliable method in hemodynamically unstable patients.
Answers are grounded in this paper and cite their sources.
TEE planimetry yields stable aortic valve area despite acute hemodynamic shifts; supports reliability in unstable patients but leaves open prospective validation.
Background:The accuracy of flow-related changes in aortic valve area (AVA) determined by the Gorlin formula or the continuity equation remains disputable. However, anatomic AVA can be determined by using by direct planimetry of transesophageal echocardiography (TEE). The purpose of this study was to assess the impact of changes in flow on AVA determined by TEE using direct planimetry. Method:Determination of AVA by TEE using direct planimetey was performed intraoperatively under three different hemodynamic conditions –pre-dobutamine (baseline) period, post-dobutamine period, post-CABG period– in 17 CABG patients and cardiac output (CO) with left ventricular ejection fraction (EF) were also determined by TEE simutaneously. The changes in aortic flow were induced by dobutamine infusion. Results:AVA at predobumaine period, post-dobutamine period, and post-CABG period were 2.99±0.80 cm 2, 3.01±0.79 cm, and 3.01±0.80 cm, respectively. Before dobutamin infusion, CO and EF were 2.01±0.64 L/min and 47± 10%. After dobutamine infusion, CO and EF were 3.03±1.05 L/min, 54±9% respectively and significantly increased by 54%, 18% than those measured before dobutamine infusion (p<0.01, p<0.01), respectively. After CABG, CO and EF were 3.86±1.86 L/min and 58±11% and also significantly increased by 98%, 26% than those measured before dobutamine infusion (p<0.01, p<0.01), respectively. However, despite of these significant hemodynamic changes, there were no significant changes in AVA and no significant correlations between these hemodynamic and AVA changes, neither at post-dobutamine period nor post-CABG period. Conclusion:The acute changes in CO and EF do not result in significant alterations in the anatomic AVA 논문접수일:1999년 10월 15일 심사완료일:2000년 7월 15일 교신저자:김종진, 130-022 서울 동대문구 전농동 620-56 가톨릭대학교 의과대학 내과학교실 전화:(02) 958-2388·전송:(02) 968-7250 E-mail:cjkim@sph.cuk.ac.kr
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Jin et al. (2000) conducted an observational in Coronary artery disease (CABG patients) (n=17). Dobutamine infusion and CABG vs. Baseline (pre-dobutamine) was evaluated on Aortic valve area (AVA) determined by TEE using direct planimetry. Acute changes in cardiac output and ejection fraction induced by dobutamine infusion and CABG did not result in significant alterations in the anatomic aortic valve area determined by transesophageal echocardiography using direct planimetry.
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