Staphylococcus aureus toxins function as superantigens by interacting with specific V beta elements of the human T-cell alpha beta receptor.
Hypothesis-generating for S. aureus pathogenesis; should not change clinical practice without human validation.
A modification of the polymerase chain reaction has been used to establish the fact that a collection of Staphylococcus aureus toxins are "superantigens," each of which interacts with the T-cell alpha beta receptor of human T cells by means of a specific set of V beta elements.
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Choi et al. (1989) studied this question.
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