Staphylococcus aureus toxins function as superantigens by interacting with specific V beta elements of the human T-cell alpha beta receptor.
A modification of the polymerase chain reaction has been used to establish the fact that a collection of Staphylococcus aureus toxins are "superantigens," each of which interacts with the T-cell alpha beta receptor of human T cells by means of a specific set of V beta elements.
Choi et al. (1989) studied this question.