What is the clinical evidence from this study?

Study design: Other. Population: Central obesity and metabolic syndrome.

What does this research mean for the field?

Central obesity independently predisposes to both atherothrombosis and venous thrombosis through a procoagulant and hypofibrinolytic state driven by inflammation, oxidative stress, dyslipidaemia, and ectopic fat. Novelty: ClaimNovelty.SYNTHESIS. Consensus alignment: ConsensusAlignment.NEUTRAL.

January 1, 2013Thrombosis and Haemostasis

Thrombosis in central obesity and metabolic syndrome: Mechanisms and epidemiology

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Key result

This document contains only the editorial board and masthead information for the journal Thrombosis and Haemostasis, with no clinical study data.

Population

Patients with central obesity and metabolic syndrome

Design

Review

Authors

Pierre‐Emmanuel MorangeVascular Medicine MAMarie‐Christine AlessiVascular Medicine

Discussion

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Implication

No clinical data provided; leaves open mechanisms linking metabolic syndrome to thrombosis.

Structured PICO

Population

Patients with central obesity and metabolic syndrome

Central obesity and metabolic syndrome drive a procoagulant state through inflammation and oxidative stress, increasing the risk of thrombotic events.

Cite This Study

Morange et al. (2013) studied Central obesity and metabolic syndrome. This document contains only the editorial board and masthead information for the journal Thrombosis and Haemostasis, with no clinical study data.

synapsesocial.com/papers/6a16d64287c007abbc052ec6 https://doi.org/10.1160/th13-01-0075

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Thrombosis in central obesity and metabolic syndrome: Mechanisms and epidemiology

Discussion

Member takes

Implication

Structured PICO

Cite This Study

Thrombosis in central obesity and metabolic syndrome: Mechanisms and epidemiology

Discussion

Member takes

Implication

Structured PICO

Cite This Study

Also Consider

Also Consider