This document contains only the editorial board and masthead information for the journal Thrombosis and Haemostasis, with no clinical study data.
Central obesity and metabolic syndrome drive a procoagulant state through inflammation and oxidative stress, increasing the risk of thrombotic events.
Central obesity is a key feature of the metabolic syndrome (metS), a multiplex risk factor for subsequent development of type 2 diabetes and cardiovascular disease. Many metabolic alterations closely related to this condition exert effects on platelets and vascular cells. A procoagulant and hypofibrinolytic state has been identified, mainly underlain by inflammation, oxidative stress, dyslipidaemia, and ectopic fat that accompany central obesity. In support of these data, central obesity independently predisposes not only to atherothrombosis but also to venous thrombosis.
Morange et al. (Tue,) conducted a other in Central obesity and metabolic syndrome. This document contains only the editorial board and masthead information for the journal Thrombosis and Haemostasis, with no clinical study data.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: