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January 30, 2003BloodOpen Access

CD4+ T-cell clones specific for wild-type factor VIII: a molecular mechanism responsible for a higher incidence of inhibitor formation in mild/moderate hemophilia A

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Authors

MJMarc JacqueminKU LeuvenVVValérie VantommeGlaxoSmithKline (Belgium)CBCécile BuhotCommissariat à l'Énergie Atomique et aux Énergies Alternatives

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Jacquemin et al. (2003) studied this question.

synapsesocial.com/papers/6a1c2cb669a4af5b15a96cdahttps://doi.org/10.1182/blood-2002-05-1369
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Mechanism and Kinetics of Factor VIII Inactivation: Study With an IgG4 Monoclonal Antibody Derived From a Hemophilia A Patient With Inhibitor1998 · 160 citations
  2. 2HLA Genotype of Patients with Severe Haemophilia A due to Intron 22 Inversion with and without Inhibitors of Factor VIII1997 · 195 citations
  3. 3Factor VIII gene inversions in severe hemophilia A: results of an international consortium study1995 · 365 citations
  4. 43-Dimensional structure of membrane-bound coagulation factor VIII: modeling of the factor VIII heterodimer within a 3-dimensional density map derived by electron crystallography2002 · 173 citations
  5. 5Two populations of Ia-like molecules on a human B cell line.1980 · 1,103 citations