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Single-cell and multi-omics analyses reveal that sepsis induces complex immune dysregulation characterized by myeloid-biased differentiation, T/NK cell exhaustion, and glial cell polarization, providing a framework for identifying novel biomarkers and precision immunotherapeutic targets. Novelty: ClaimNovelty.SYNTHESIS. Consensus alignment: ConsensusAlignment.NEUTRAL.

Immune regulation and therapeutic targets in sepsis: insights from single-cell transcriptomics

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