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January 1, 1997Journal of Biological ChemistryOpen Access

The Mitogen-activated Protein Kinase and JAK-STAT Signaling Pathways Are Required for an Oncostatin M-responsive Element-mediated Activation of Matrix Metalloproteinase 1 Gene Expression

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Authors

EKEdward KorzusUniversity of California, RiversideHNHideaki NagaseVascular MedicineRRRussell E. RydellAthena Group (United States)

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Korzus et al. (1997) studied this question.

synapsesocial.com/papers/6a1d194373c56dd1bd2f2adahttps://doi.org/10.1074/jbc.272.2.1188
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Developmental Abnormalities in Mice Transgenic for Bovine Oncostatin M1995 · 121 citations
  2. 2TRANSCRIPTIONAL RESPONSES TO POLYPEPTIDE LIGANDS: The JAK-STAT Pathway1995 · 1,832 citations
  3. 3Stat3: a STAT Family Member Activated by Tyrosine Phosphorylation in Response to Epidermal Growth Factor and Interleukin-61994 · 2,102 citations
  4. 4Proliferative responses and binding properties of hematopoietic cells transfected with low-affinity receptors for leukemia inhibitory factor, oncostatin M, and ciliary neurotrophic factor.1994 · 134 citations
  5. 5Identification of the interleukin-6/oncostation M response element in the rat tissue inhibitor of metalloproteinases-1 (TIMP-1)Promoter1995 · 110 citations