Answers are grounded in this paper and cite their sources.
This case report substantiates the primary role of A-V nodal conduction delay in the genesis of paroxysmal SVT.
In the present report, A-V dissociation with concealed retrograde conduction into the A-V node, caused SINUS beats to exhibit antegrade A-V nodal conduction delay sufficient to result in episodes of supraventricular tachycardia (SVT). In this patient, atrial ectopic premature depolarizations were never observed. The primary role of A-V nodal conduction delay in the genesis of paroxysmal SVT is substantiated by this unusual electrophysiological observation.
No takes yet. Share an insight, caveat, or question.
Goldreyer et al. (1971) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: