Key result
Direct current cardioversion for paroxysmal atrial flutter caused pulmonary edema in a 68-year-old man, likely due to transient left ventricular diastolic dysfunction from the shock.
Why the study?
Can direct current cardioversion cause pulmonary edema in patients with paroxysmal atrial flutter?
Case Report (n=1)
Can direct current cardioversion cause pulmonary edema in patients with paroxysmal atrial flutter?
Direct current cardioversion can rarely cause transient left ventricular diastolic dysfunction leading to pulmonary edema, even in patients without underlying organic heart disease.
Answers are grounded in this paper and cite their sources.
DC cardioversion may rarely trigger pulmonary edema via transient diastolic dysfunction; hypothesis-generating case report requiring prospective confirmation.
This report describes a patient with the pulmonary edema after cardioversion for paroxysmal atrial flutter without organic heart disease. A 68-year-old man was admitted to hospital for paroxysmal atrial flutter. Antiarrhythmic agents were not effective, and direct current cardioversion was performed on the 4th hospital day. Three hours after cardioversion, the patient complained of dyspnea, and a chest X-ray showed pulmonary edema. He responded to oxygen, intravenous furosemide and drip infusion of nitroglycerine. During tapering of the medication, his condition remained stable. The patient was discharged on the 7th day after admission. Echocardiographic findings indicated that transient left ventricular diastolic dysfunction due to direct current shock was the most likely cause of the lung edema.
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Kobayashi et al. (2000) conducted a case report in Paroxysmal atrial flutter (n=1). Direct current cardioversion was evaluated on Pulmonary edema. Direct current cardioversion for paroxysmal atrial flutter caused pulmonary edema in a 68-year-old man, likely due to transient left ventricular diastolic dysfunction from the shock.
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