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February 27, 2012PLoS ONEOpen Access

Preventing Phosphorylation of Sterol Regulatory Element-Binding Protein 1a by MAP-Kinases Protects Mice from Fatty Liver and Visceral Obesity

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Authors

JKJörg KotzkaBKBirgit KnebelJHJutta Haas

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Kotzka et al. (2012) studied this question.

synapsesocial.com/papers/6a201d23d47ed904550dc5eehttps://doi.org/10.1371/journal.pone.0032609
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Isoform 1c of sterol regulatory element binding protein is less active than isoform 1a in livers of transgenic mice and in cultured cells.1997 · 838 citations
  2. 2Cholesterol feeding reduces nuclear forms of sterol regulatory element binding proteins in hamster liver1997 · 154 citations
  3. 3Pro-Leu-Ser/Thr-Pro is a consensus primary sequence for substrate protein phosphorylation. Characterization of the phosphorylation of c-myc and c-jun proteins by an epidermal growth factor receptor threonine 669 protein kinase1991 · 481 citations
  4. 4Growth Factor-induced Phosphorylation of Sterol Regulatory Element-binding Proteins Inhibits Sumoylation, Thereby Stimulating the Expression of Their Target Genes, Low Density Lipoprotein Uptake, and Lipid Synthesis2008 · 82 citations