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During the past ten years clinical experience has indicated that bacterial interference can be useful in curtailing staphylococcal disease among newborn babies in the nursery during epidemics and in patients with chronic furunculosis. Studies in a variety of animal models and in vitro have permitted investigations of mechanisms responsible for this ecologic relationship. However, to date, the explanation of this phenomenon in the human remains unknown.
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Henry R. Shinefield (1971) studied this question.
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