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January 1, 1998AJP Heart and Circulatory Physiology

Endothelial and smooth muscle cell conduction in arterioles controlling blood flow

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Population

Arterioles (diameter 47 ± 1 μm, n = 63) supplying blood flow to the cheek pouch of anesthetized hamsters

Comparison

Micropipette delivery of norepinephrine… vs Resting state / baseline

Design

Preclinical

Key result

Micropipette delivery of norepinephrine or phenylephrine produced smooth muscle cell depolarization (5–41 mV) and vasoconstriction (7–49 μm) independent of the endothelium.

Authors

DWDonald G. WelshWestern UniversitySSSteven S. SegalVascular / Pulmonary Vascular

Discussion

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Overview

Supports distinct smooth muscle conduction for adrenergic constriction in arterioles; leaves open translation to human microvascular regulation.

Structured PICO

P
Population
63 arterioles from the cheek pouch of anesthetized hamsters were studied to investigate cellular pathways of constriction and dilation.
I
Intervention
Micropipette delivery of norepinephrine (NE), phenylephrine (PE), KCl, or acetylcholine
C
Comparator
Resting state / baseline
O
Outcome
Membrane potential (Em) changes and vasoconstriction/vasodilationsurrogate

Depolarization and constriction conduct along smooth muscle independently of the endothelium, whereas vasodilation conducts via hyperpolarization along homologously coupled cells, indicating these pathways are not electrically coupled during blood flow control.

Cite This Study

Welsh et al. (1998) studied this question. Micropipette delivery of vasoactive agents (NE, PE, KCl, Acetylcholine) was evaluated on Membrane potential and vessel diameter changes. Micropipette delivery of norepinephrine or phenylephrine produced smooth muscle cell depolarization (5–41 mV) and vasoconstriction (7–49 μm) independent of the endothelium.

synapsesocial.com/papers/6a2304eec650520b07cb27echttps://doi.org/10.1152/ajpheart.1998.274.1.h178
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