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Significance Jasmonates are indispensable for plant growth, as well as for adaptation to biotic and abiotic stresses. The synthesis of jasmonates is initiated in the chloroplast, where the precursor, 12-oxophytodienoic acid (OPDA) is derived from plastid lipids. Despite the fact that much information has been gained on the enzymes involved in the biosynthesis and signaling pathways of jasmonates, the export of OPDA from plastids remains enigmatic. In the present study, we showed that JASSY, a protein localized to the outer chloroplast envelope, facilitates export of OPDA from the chloroplast. Loss-of-function mutants lead to abolished jasmonate accumulation, which in turn results in increased susceptibility to cold treatment, as well as pathogen attack.
Guan et al. (Wed,) studied this question.