Demonstrates restoration of sensory input in corneal neurotization, suggesting improved ocular health.
Neurotrophic keratopathy (NK) results from loss of trigeminal innervation to the cornea, disrupting epithelial integrity, tear film regulation, and ocular surface homeostasis. Conventional treatments address the surface consequences of denervation but do not restore sensory input. Corneal neurotization (CN) directly addresses this biological deficit by transferring healthy donor axons to the anesthetic cornea, re-establishing afferent innervation and creating the conditions for epithelial maintenance and visual rehabilitation.
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Ashton et al. (2026) studied this question.
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